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leprdb db b6 bks d leprdb j mice  (Jackson Laboratory)

 
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    Jackson Laboratory leprdb db b6 bks d leprdb j mice
    Leprdb Db B6 Bks D Leprdb J Mice, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/b6+bks+d+leprdb+j+db+mice/bks+cg+dock7+m/pm42033177-52-10-16
    Average 86 stars, based on 1 article reviews
    leprdb db b6 bks d leprdb j mice - by Bioz Stars, 2026-10
    86/100 stars

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    other:

    Article Title: Marine n-3 fatty acids protect against renal function deterioration in diabetic patients by modulating lipoproteins and promoting cholesterol efflux.
    Article Snippet: The fat-1 mice used as parental stock were kindly provided by Professor Yi-Fan Dai from Nanjing Medical University, and the B6.BKS(D)-Leprdb/J (db/+) mice were obtained from Jackson Laboratory (Bar Harbor, Maine, USA).



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    Loss of spinophilin significantly reduces weight gain in lean and two mouse models of obesity. A and B : male ( A ) or female ( B ) <t>Lepr</t> +/+ / Spino +/+ , Lepr db/db / Spino +/+ , Lepr +/+ / Spino −/− , and Lepr db/db / Spino −/− mice weights were taken bi-weekly from 4 to 20 wk and plotted. A mixed-effects three-way ANOVA was performed initially, followed by a two-way ANOVA to determine the effect of spinophilin genotype, age, and an interaction within the Lepr +/+ and Lepr db/db genotypes individually. C and D : male ( C ) and female ( D ) Spino +/+ /high-fat diet-fed (HFF) and Spino −/− /HFF mice were weighed bi-weekly and plotted. For two-way ANOVAs, significant spinophilin genotype, time, and interaction (spinophilin genotype × time) are shown. n = 4–12 mice per group for each age point. Data ± SE are shown. * P < 0.05, ** P < 0.01, *** P < 0.001, **** P < 0.001. All statistics are shown in Supplemental Material ( https://doi.org/10.6084/m9.figshare.22507135.v1 ).
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    Loss of spinophilin significantly reduces weight gain in lean and two mouse models of obesity. A and B : male ( A ) or female ( B ) Lepr +/+ / Spino +/+ , Lepr db/db / Spino +/+ , Lepr +/+ / Spino −/− , and Lepr db/db / Spino −/− mice weights were taken bi-weekly from 4 to 20 wk and plotted. A mixed-effects three-way ANOVA was performed initially, followed by a two-way ANOVA to determine the effect of spinophilin genotype, age, and an interaction within the Lepr +/+ and Lepr db/db genotypes individually. C and D : male ( C ) and female ( D ) Spino +/+ /high-fat diet-fed (HFF) and Spino −/− /HFF mice were weighed bi-weekly and plotted. For two-way ANOVAs, significant spinophilin genotype, time, and interaction (spinophilin genotype × time) are shown. n = 4–12 mice per group for each age point. Data ± SE are shown. * P < 0.05, ** P < 0.01, *** P < 0.001, **** P < 0.001. All statistics are shown in Supplemental Material ( https://doi.org/10.6084/m9.figshare.22507135.v1 ).

    Journal: American Journal of Physiology - Endocrinology and Metabolism

    Article Title: Mechanisms of spinophilin-dependent pancreas dysregulation in obesity

    doi: 10.1152/ajpendo.00099.2023

    Figure Lengend Snippet: Loss of spinophilin significantly reduces weight gain in lean and two mouse models of obesity. A and B : male ( A ) or female ( B ) Lepr +/+ / Spino +/+ , Lepr db/db / Spino +/+ , Lepr +/+ / Spino −/− , and Lepr db/db / Spino −/− mice weights were taken bi-weekly from 4 to 20 wk and plotted. A mixed-effects three-way ANOVA was performed initially, followed by a two-way ANOVA to determine the effect of spinophilin genotype, age, and an interaction within the Lepr +/+ and Lepr db/db genotypes individually. C and D : male ( C ) and female ( D ) Spino +/+ /high-fat diet-fed (HFF) and Spino −/− /HFF mice were weighed bi-weekly and plotted. For two-way ANOVAs, significant spinophilin genotype, time, and interaction (spinophilin genotype × time) are shown. n = 4–12 mice per group for each age point. Data ± SE are shown. * P < 0.05, ** P < 0.01, *** P < 0.001, **** P < 0.001. All statistics are shown in Supplemental Material ( https://doi.org/10.6084/m9.figshare.22507135.v1 ).

    Article Snippet: Male and Female Lepr db/+ mice [B6.BKS(D)-Leprdb/J, Stock No. 000697, Jackson Laboratories, Bar Harbor, ME] and whole body heterozygous spinophilin mice (Stock No. 018609; RRID: MMRRC_049172-UCD) were initially purchased from Jackson Laboratories and breeding colonies were maintained at IUPUI.

    Techniques:

    Spinophilin protein interactions with protein phosphatase 1 (PP1), neurabin, and myosin-9 in wild-type (WT), Ins2 Akita , and Lepr db/db mice. A : spinophilin was immunoprecipitated from pancreas of adult WT, Ins2 Akita (AK), and Lepr db/db (DB) mice. Immunoblotting for spinophilin, PP1α, myosin-9, and neurabin was performed. B and C : PP1α ( B ) or neurabin ( C ) expression in spinophilin immunoprecipitates was normalized to spinophilin expression in the immuno precipitate. A normalized ratio was plotted. n = 3–5 mice per group. ** P < 0.01. All statistics are shown in Supplemental Material ( https://doi.org/10.6084/m9.figshare.22507135.v1 ).

    Journal: American Journal of Physiology - Endocrinology and Metabolism

    Article Title: Mechanisms of spinophilin-dependent pancreas dysregulation in obesity

    doi: 10.1152/ajpendo.00099.2023

    Figure Lengend Snippet: Spinophilin protein interactions with protein phosphatase 1 (PP1), neurabin, and myosin-9 in wild-type (WT), Ins2 Akita , and Lepr db/db mice. A : spinophilin was immunoprecipitated from pancreas of adult WT, Ins2 Akita (AK), and Lepr db/db (DB) mice. Immunoblotting for spinophilin, PP1α, myosin-9, and neurabin was performed. B and C : PP1α ( B ) or neurabin ( C ) expression in spinophilin immunoprecipitates was normalized to spinophilin expression in the immuno precipitate. A normalized ratio was plotted. n = 3–5 mice per group. ** P < 0.01. All statistics are shown in Supplemental Material ( https://doi.org/10.6084/m9.figshare.22507135.v1 ).

    Article Snippet: Male and Female Lepr db/+ mice [B6.BKS(D)-Leprdb/J, Stock No. 000697, Jackson Laboratories, Bar Harbor, ME] and whole body heterozygous spinophilin mice (Stock No. 018609; RRID: MMRRC_049172-UCD) were initially purchased from Jackson Laboratories and breeding colonies were maintained at IUPUI.

    Techniques: Immunoprecipitation, Western Blot, Expressing